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zsgreen hanbio technology n a ckap4 oe lentivirus genechem n a lentiviral negative control genechem n a software  (Genechem)

 
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    Genechem zsgreen hanbio technology n a ckap4 oe lentivirus genechem n a lentiviral negative control genechem n a software
    Zsgreen Hanbio Technology N A Ckap4 Oe Lentivirus Genechem N A Lentiviral Negative Control Genechem N A Software, supplied by Genechem, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/negative+control+lentiviruses/pm42207635-298-55-61?v=Genechem
    Average 86 stars, based on 1 article reviews
    zsgreen hanbio technology n a ckap4 oe lentivirus genechem n a lentiviral negative control genechem n a software - by Bioz Stars, 2026-08
    86/100 stars

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    The effects of targeted MALAT1 on proinflammatory cytokines, IA formation, VSMC transformation, and the Smad pathway in IA rats. Effects of coinjection of MALAT1-silenced and SEMA3C-overexpressing <t>lentiviruses</t> on relative MALAT1 expression in IA tissue (A) , serum TNF-α (B) , serum IL-1β (C) , and serum IL-6 levels (D) , histological changes (E) , SEMA3C IHC scores (F) , α-SMA IHC scores, OPN IHC scores, MMP2 IHC scores, MMP9 IHC scores (G) , p-smad2 IHC scores and p-smad3 IHC scores (H) . * P < 0.05, ** P < 0.01, *** P < 0.001.
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    The effects of targeted MALAT1 on proinflammatory cytokines, IA formation, VSMC transformation, and the Smad pathway in IA rats. Effects of coinjection of MALAT1-silenced and SEMA3C-overexpressing lentiviruses on relative MALAT1 expression in IA tissue (A) , serum TNF-α (B) , serum IL-1β (C) , and serum IL-6 levels (D) , histological changes (E) , SEMA3C IHC scores (F) , α-SMA IHC scores, OPN IHC scores, MMP2 IHC scores, MMP9 IHC scores (G) , p-smad2 IHC scores and p-smad3 IHC scores (H) . * P < 0.05, ** P < 0.01, *** P < 0.001.

    Journal: Frontiers in Cellular Neuroscience

    Article Title: Silencing MALAT1 represses pathological progression, inflammation, and vascular smooth muscle cell phenotype switching by regulating the SEMA3C-mediated Smad pathway in intracranial aneurysms

    doi: 10.3389/fncel.2026.1706518

    Figure Lengend Snippet: The effects of targeted MALAT1 on proinflammatory cytokines, IA formation, VSMC transformation, and the Smad pathway in IA rats. Effects of coinjection of MALAT1-silenced and SEMA3C-overexpressing lentiviruses on relative MALAT1 expression in IA tissue (A) , serum TNF-α (B) , serum IL-1β (C) , and serum IL-6 levels (D) , histological changes (E) , SEMA3C IHC scores (F) , α-SMA IHC scores, OPN IHC scores, MMP2 IHC scores, MMP9 IHC scores (G) , p-smad2 IHC scores and p-smad3 IHC scores (H) . * P < 0.05, ** P < 0.01, *** P < 0.001.

    Article Snippet: MALAT1 overexpression (oeMALAT1) lentivirus (Genechem, China), negative control overexpression (oeNC) lentivirus (Genechem, China), MALAT1 short hairpin (shMALAT1) lentivirus (Genechem, China) and negative control short hairpin (shNC) lentivirus (Genechem, China) were used to infect the VSMCs at a multiplicity of infection (MOI) of 20:1.

    Techniques: Transformation Assay, Expressing